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Molecular networks underlying Treg suppressor function
Treg dysregulation is key in various pathologic conditions such as in autoimmune diseases, allergic diseases, infectious diseases, cancer, neurodegenerative diseases and metabolic inflammation.
Human individuals show great variance in both innate and adaptive immune responses following immune stimuli. However, little is known about which molecular subnetworks quantitatively control the resp
This project will experimentally study whether and how critical transitions occur in the immune system and whether early warning molecular signatures can be used for potential therapeutic intervention
